Introduction Critical Care Nursing 7th Edition By Sole Klein
Introduction Critical Care Nursing 7th Edition By Sole Klein
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Chapter 21: Burns
Complete Chapter Questions With Answers
Sample Questions Are Posted Below
MULTIPLE CHOICE
| a. | blood urea nitrogen. |
| b. | daily weight. |
| c. | hourly intake and urine output. |
| d. | serum potassium. |
ANS: C
During initial fluid resuscitation, urine output helps guide fluid resuscitation needs. Measuring hourly intake and output is most effective in determining the needs for additional fluid infusion than is urine output alone. Blood urea nitrogen may be used to monitor volume status, but it is affected by the hypermetabolic state seen after burns, so it is not the optimal measure of intravascular fluid status. Daily weight measures overall volume status, not just intravascular volume. Serum potassium is released with tissue damage and thus is not the optimum measure of intravascular fluid status.
DIF: Cognitive Level: Remember/Knowledge REF: Box 21-1
OBJ: Discuss the primary and secondary survey assessments during resuscitation and the acute phases of burn management. TOP: Nursing Process Step: Assessment
MSC: NCLEX Client Needs Category: Physiological Integrity
| a. | catecholamine-induced vasoconstriction. |
| b. | decreased glomerular filtration. |
| c. | increased capillary permeability. |
| d. | loss of integument barrier. |
ANS: C
Capillary permeability is altered in burns beyond the area of tissue damage, resulting in significant shift of proteins, fluid, and electrolytes resulting in edema (third-spacing). Catecholamine-induced vasoconstriction does not produce edema. Decreased glomerular filtration may cause fluid retention, but it is not responsible for the extensive edema seen after burn injury. Loss of integument barrier does not cause edema.
DIF: Cognitive Level: Remember/Knowledge
REF: p. 592 | Table 21-4 OBJ: Describe the pathophysiology of burns.
TOP: Nursing Process Step: Evaluation
MSC: NCLEX Client Needs Category: Physiological Integrity
| a. | acute kidney injury. |
| b. | acute respiratory distress syndrome. |
| c. | infection. |
| d. | stress ulcers. |
ANS: C
The loss of skin as the primary barrier against microorganisms and activation of the inflammatory response cascades results in immunosuppression, placing the patient at an increased risk of infection. A systemic inflammatory response syndrome (SIRS) also increases the risk of acute kidney injury in the presence of poor tissue perfusion. Acute respiratory distress syndrome is also a potential complication, but the risk of infection is greater because of the loss of the skin barrier. Catecholamine release and gastrointestinal ischemia are the causes of stress ulcers.
DIF: Cognitive Level: Remember/Knowledge REF: pp. 593-594
OBJ: Describe the pathophysiology of burns.
TOP: Nursing Process Step: Evaluation
MSC: NCLEX Client Needs Category: Physiological Integrity
| a. | 2800 mL |
| b. | 7000 mL |
| c. | 14 L |
| d. | 28 L |
ANS: C
154 pounds/2.2 = 70 kg
4 ´ 70 kg ´ 50 = 14,000 mL, or 14 liters.
DIF: Cognitive Level: Apply/Application REF: p. 603 | Box 21-1
OBJ: Formulate a plan of care for the patient with a burn injury.
TOP: Nursing Process Step: Planning
MSC: NCLEX Client Needs Category: Physiological Integrity
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