Lehne's Pharmacology for Nursing Care 9th Edition by Jacqueline Burchum
Lehne's Pharmacology for Nursing Care 9th Edition by Jacqueline Burchum
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Chapter 48: Drugs for Heart Failure
Complete Chapter Questions With Answers
Sample Questions Are Posted Below
MULTIPLE CHOICE
| a. | hypernatremia. |
| b. | hypertension. |
| c. | hyperkalemia. |
| d. | hypokalemia. |
ANS: C
One of the principal effects of angiotensin-converting enzyme (ACE) inhibitors is hyperkalemia, which is due to decreased aldosterone release arising from blockage of angiotensin II. There is no indication that careful monitoring of sodium for increased levels is indicated. Vasotec is indicated for heart failure, not hypertension. The drug therapy should be monitored to ascertain its effectiveness, but hyperkalemia is the main concern. Hyperkalemia, not hypokalemia, is a concern because of the decreased aldosterone release that occurs with blockage of angiotensin II.
PTS: 1 DIF: Cognitive Level: Application REF: p. 520
TOP: Nursing Process: Planning
MSC: NCLEX Client Needs Category: Physiologic Integrity: Reduction of Risk Potential
| a. | “An increase in arteriolar tone to improve tissue perfusion can decrease resistance.” |
| b. | “An increase in contractility to increase cardiac output can cause pulmonary edema.” |
| c. | “When the heart rate increases to increase cardiac output, it can prevent adequate filling of the ventricles.” |
| d. | “When venous tone increases to increase ventricular filling, an increase in arterial pressure occurs.” |
ANS: C
The heart rate increases to improve cardiac output, but it may prevent adequate ventricular filling. An increase in arteriole tone improves tissue perfusion but also increases both the resistance to the pumping of the heart and the cardiac workload. Increased contractility helps improve cardiac output but is detrimental because it increases the oxygen demand of the heart. An increase in venous tone improves ventricular filling, but as the ventricles fail, blood can back up and cause pulmonary edema.
PTS: 1 DIF: Cognitive Level: Analysis REF: p. 518
TOP: Nursing Process: Assessment
MSC: NCLEX Client Needs Category: Physiologic Integrity: Physiologic Adaptation
| a. | ACE inhibitor |
| b. | Digoxin [Lanoxin] |
| c. | Furosemide [Lasix] |
| d. | Spironolactone [Aldactone] |
ANS: C
This patient shows signs of fluid volume overload and needs a diuretic. Furosemide is a loop diuretic, which can produce profound diuresis very quickly even when the glomerular filtration rate (GFR) is low. An ACE inhibitor will not reduce fluid volume overload. Digoxin has a positive inotropic effect on the heart, which may improve renal perfusion, but this is not its primary effect. Spironolactone is a potassium-sparing diuretic with weak diuresis effects; it is used in conjunction with other diuretics to improve electrolyte balance.
PTS: 1 DIF: Cognitive Level: Application REF: pp. 517-518 | pp. 519-522
TOP: Nursing Process: Implementation
MSC: NCLEX Client Needs Category: Physiologic Integrity: Reduction of Risk Potential
| a. | decreased urine output. |
| b. | increased blood pressure. |
| c. | jugular vein distension. |
| d. | shortness of breath. |
ANS: A
As the heart rate increases, ventricular filling decreases, and cardiac output and renal perfusion decrease. Tachycardia does not elevate blood pressure. Jugular vein distension and shortness of breath occur with fluid volume overload.
PTS: 1 DIF: Cognitive Level: Analysis REF: p. 518
TOP: Nursing Process: Assessment
MSC: NCLEX Client Needs Category: Physiologic Integrity: Physiologic Adaptation
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