Chapter 48: Shock, Multiple Organ Dysfunction Syndrome, and Burns in Adults

Pathophysiology The Biologic Basis for Disease in Adults and Children, 7th Edition by Kathryn L.

$2.99

Chapter 48: Shock, Multiple Organ Dysfunction Syndrome, and Burns in Adults

 

Complete Chapter Questions With Answers

 

Sample Questions Are Posted Below

 

MULTIPLE CHOICE

 

  1. What is the final outcome of impaired cellular metabolism?
a. Cellular alterations in the heart and brain
b. Buildup of cellular waste products
c. Cellular alterations in the vasculature structures and kidneys
d. Impairment of urine excretion

 

 

ANS:  B

The common pathway in all types of shock is impairment of cellular metabolism as a result of decreased delivery of oxygen and nutrients, which are frequently coupled with an increased demand, the consumption of oxygen and nutrients, and a decreased removal of cellular waste products. Of the options available, this selection is the only accurate outcome.

 

PTS:   1                    REF:   Page 1669

 

  1. Which clinical manifestation of septic shock confirms an elevation in immune system response?
a. Tachycardia c. Low respiratory rate
b. Increased white blood cell count d. Hypothermia

 

 

ANS:  B

Clinical manifestations common in septic shock are fever, high heart rate, high respiratory rate, or elevations in immune responses, such as increased white blood cells and circulating blood glucose.

 

PTS:   1                    REF:   Page 1671

 

  1. The release of catecholamine by the adrenal glands compensate for which initial effects of hypovolemic shock?
a. Interstitial fluid moves out of the vascular compartment.
b. Systemic vascular resistance is decreased.
c. Heart rate is increased.
d. Water excretion is increased.

 

 

ANS:  C

Compensatory mechanisms (see Figure 48-3) initially offset hypovolemia. Heart rate and systemic vascular resistance increase as a result of catecholamine release by the adrenal glands, which boosts cardiac output and tissue perfusion pressures. Compelled by a decrease in capillary hydrostatic pressures, interstitial fluid moves into the vascular compartment. The liver and spleen add to blood volume by disgorging stored red blood cells and plasma. In the kidneys, renin (through several intermediaries) stimulates aldosterone release and the retention of sodium and therefore water, whereas antidiuretic hormone (ADH), or vasopressin, from the posterior pituitary gland increases water retention. Data on the compensation of ADH, however, show that as shock worsens, ADH in plasma decreases.

 

PTS:   1                    REF:   Page 1672

 

  1. Hypovolemic shock begins to develop when intravascular volume has decreased by what percentage?
a. 5 c. 15
b. 10 d. 20

 

 

ANS:  C

Hypovolemic shock begins to develop when intravascular volume has decreased by approximately 15%.

 

PTS:   1                    REF:   Page 1672

 

  1. What type of shock develops as a result of the overstimulation of the parasympathetic nervous system or the understimulation of the sympathetic nervous system?
a. Septic c. Anaphylactic
b. Cardiogenic d. Vasogenic

 

 

ANS:  D

Only vasogenic shock refers to a widespread and massive vasodilation resulting from an imbalance between parasympathetic and sympathetic stimulation of vascular smooth muscle.

 

PTS:   1                    REF:   Pages 1673-1674

Additional information

Add Review

Your email address will not be published. Required fields are marked *